Do you know how to get rid of fruit flies? Fruit flies are extremely hard to get rid of and removal of an infestation can be very difficult, as larvae continue to hatch in fruit even as the adult population is gone. Many people make smelly apple cider vinegar traps all over their home, some dangle sticky fly strips all over the house while some smash them with fly swatters.
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A great feature our bug zapper comes with, is the ability to bait and attract fruit flies from all over your house. Attach our bait tray to the bug zapper, and place fruit or anything youd like to use as their favorite bait. When the flies are attracted, you will hear a pop and crackle sound, and they will simply fall into the catch tray. Very clean, safe and easy way to handle getting rid of all the fruit flies in your home without having to use sticky and smelly traps.
Drosophila melanogaster is a species of fly (the
taxonomic order Diptera) in the family Drosophilidae. The species is known
generally as the common fruit fly or vinegar fly. Starting with Charles W.
Woodworth's proposal of the use of this species as a model organism, D.
melanogaster continues to be widely used for biological research in studies of
genetics, physiology, microbial pathogenesis, and life history evolution. It is
typically used because it is an animal species that is easy to care for, has
four pairs of chromosomes, breeds quickly, and lays many eggs. D. melanogaster
is a common pest in homes, restaurants, and other occupied places where food is
Flies belonging to the family Tephritidae are also called "fruit flies". This can cause confusion, especially in Australia and South Africa, where the Mediterranean fruit fly Ceratitis capitata is an economic pest.
Wildtype fruit flies are yellow-brown, with brick-red eyes and transverse black rings across the abdomen. They exhibit sexual dimorphism: females are about 2.5 millimeters (0.098 in) long; males are slightly smaller with darker backs. Males are easily distinguished from females based on colour differences, with a distinct black patch at the abdomen, less noticeable in recently emerged flies, and the sexcombs (a row of dark bristles on the tarsus of the first leg). Furthermore, males have a cluster of spiky hairs (claspers) surrounding the reproducing parts used to attach to the female during mating. There are extensive images at FlyBase.
The D. melanogaster lifespan is about 30 days at 29 C
(84 F). It had been recorded that their lifespan can be increased to 3 months.
The developmental period for D. melanogaster varies with temperature, as with many ectothermic species. The shortest development time (egg to adult), 7 days, is achieved at 28 C (82 F). Development times increase at higher temperatures (11 days at 30 C or 86 F) due to heat stress. Under ideal conditions, the development time at 25 C (77 F) is 8.5 days, at 18 C (64 F) it takes 19 days and at 12 C (54 F) it takes over 50 days. Under crowded conditions, development time increases, while the emerging flies are smaller. Females lay some 400 eggs (embryos), about five at a time, into rotting fruit or other suitable material such as decaying mushrooms and sap fluxes. The eggs, which are about 0.5 mm long, hatch after 12-15 hours (at 25 C or 77 F). The resulting larvae grow for about 4 days (at 25 C) while molting twice (into second- and third-instar larvae), at about 24 and 48 h after hatching. During this time, they feed on the microorganisms that decompose the fruit, as well as on the sugar of the fruit itself. The mother put feces on the egg sacs to establish the same microbial composition in the larvaes guts which has worked positive for herself. Then the larvae encapsulate in the puparium and undergo a four-day-long metamorphosis (at 25 C), after which the adults eclose (emerge).
Mating fruit flies: Note the sex combs on the forelegs of the male
Females become receptive to courting males at about 8-12 hours after emergence. Specific neuron groups in females have been found to affect copulation behavior and mate choice. One such group in the abdominal nerve cord allows the female fly to pause her body movement to copulate. Activation of these neurons induces the female to cease movement and orient herself towards the male to allow for mounting. If the group is inactivated, the female remains in motion and does not copulate. Various chemical signals such as male pheromones often are able to activate the group.
The female fruit fly prefers a shorter duration when it comes to sex. Males, on the other hand, prefer it to last longer. Males perform a sequence of five behavioral patterns to court females. First, males orient themselves while playing a courtship song by horizontally extending and vibrating their wings. Soon after, the male positions itself at the rear of the female's abdomen in a low posture to tap and lick the female genitalia. Finally, the male curls its abdomen, and attempts copulation. Females can reject males by moving away, kicking, and extruding their ovipositor. Copulation lasts around 15-20 minutes, during which males transfer a few hundred, very long (1.76 mm) sperm cells in seminal fluid to the female. Females store the sperm in a tubular receptacle and in two mushroom-shaped spermathecae; sperm from multiple matings compete for fertilization. A last male precedence is believed to exist in which the last male to mate with a female sires about 80% of her offspring. This precedence was found to occur through displacement and incapacitation. The displacement is attributed to sperm handling by the female fly as multiple matings are conducted and is most significant during the first 1-2 days after copulation. Displacement from the seminal receptacle is more significant than displacement from the spermathecae. Incapacitation of first male sperm by second male sperm becomes significant 2-7 days after copulation. The seminal fluid of the second male is believed to be responsible for this incapacitation mechanism (without removal of first male sperm) which takes effect before fertilization occurs. The delay in effectiveness of the incapacitation mechanism is believed to be a protective mechanism that prevents a male fly from incapacitating its own sperm should it mate with the same female fly repetitively. Sensory neurons in the uterus of female D. melanogaster respond to a male protein, sex peptide, which is found in sperm. This protein makes the female reluctant to copulate for about 10 days after insemination. The signal pathway leading to this change in behavior has been determined. The signal is sent to a brain region that is a homolog of the hypothalamus and the hypothalamus then controls sexual behavior and desire
D. melanogaster is often used for life extension studies. Beginning in 1980, Michael R. Rose conducted a groundbreaking study in experimental evolution resulting in "Methuselah" flies which had roughly double the lifespan of a wild-type fruit fly. More recently, particular genes such as the INDY gene have been identified which are purported to increase lifespan when mutated.
Behavior refers to the actions an organism might take
in response to various internal or external inputs. Changes in behavior may
indicate learning, which is when an organisms adapts to a situation or
phenomenon by changing a particular behavioral response. Learning is usually
associated with an increase in fitness, especially when the adapted behavior is
an aspect of sexual behavior. D. melanogaster males exhibit a strong
reproductive learning curve. That is, with sexual experience, these flies tend
to modify their future mating behavior in multiple ways. These changes include
increased selectivity for courting only intraspecifically, as well as decreased
courtship times. Sexually nave D. melanogaster males are known to spend
significant time courting interspecifically, such as with D. simulans flies.
Nave D. melanogaster will also attempt to court females that are not yet
sexually mature and other males. D. melanogaster males show little to no
preference for D. melanogaster females over females of other species or even
other male flies. However, after D. simulans or other flies incapable of
copulation have rejected the males advances, D. melanogaster males are much
less likely to spend time courting nonspecifically in the future. This apparent
learned behavior modification seems to be evolutionarily significant, as it
allows the males to avoid investing energy into hopeless sexual encounters.
In addition, males with previous sexual experience will modify their courtship dance when attempting to mate with new females the experienced males spend less time courting and therefore have lower mating latencies, meaning that they are able to reproduce more quickly. This decreased mating latency leads to a greater mating efficiency for experienced males over nave males. This modification also appears to have obvious evolutionary advantages, as increased mating efficiency is extremely important in the eyes of natural selection.
D. melanogaster was among the first organisms used for
genetic analysis, and today it is one of the most widely used and genetically
best-known of all eukaryotic organisms. All organisms use common genetic
systems; therefore, comprehending processes such as transcription and
replication in fruit flies helps in understanding these processes in other
eukaryotes, including humans.
Charles W. Woodworth is credited with being the first to breed Drosophila in quantity and for suggesting to W. E. Castle that they might be used for genetic research during his time at Harvard University.
Thomas Hunt Morgan began using fruit flies in experimental studies of heredity at Columbia University in 1910. His laboratory was located on the top floor of Schermerhorn Hall, which became known as the Fly Room. The Fly Room was cramped with eight desks, each occupied by students and their experiments. They started off experiments using milk bottles to rear the fruit flies and handheld lenses for observing their traits. The lenses were later replaced by microscopes, which enhanced their observations. The Fly Room was the source of some of the most important research in the history of biology. Morgan and his students eventually elucidated many basic principles of heredity, including sex-linked inheritance, epistasis, multiple alleles, and gene mapping.
"Thomas Hunt Morgan and colleagues extended Mendel's work by describing X-linked inheritance and by showing that genes located on the same chromosome do not show independent assortment. Studies of X-linked traits helped confirm that genes are found on chromosomes, while studies of linked traits led to the first maps showing the locations of genetic loci on chromosomes". The first maps of Drosophila chromosomes were completed by Alfred Sturtevant.
D. melanogaster is one of the most studied organisms
in biological research, particularly in genetics and developmental biology. The
several reasons include:
Its care and culture requires little equipment and uses little space even when using large cultures, and the overall cost is low.
It is small and easy to grow in the laboratory and its morphology is easy to identify once anesthetized (usually with ether, carbon dioxide gas, by cooling, or with products like FlyNap).
It has a short generation time (about 10 days at room temperature), so several generations can be studied within a few weeks.
It has a high fecundity (females lay up to 100 eggs per day, and perhaps 2000 in a lifetime).
Males and females are readily distinguished and virgin females are easily isolated, facilitating genetic crossing.
The mature larvae show giant chromosomes in the salivary glands called polytene chromosomes "puffs" indicate regions of transcription and hence gene activity.
It has only four pairs of chromosomes: three autosomes, and one sex chromosome.
Males do not show meiotic recombination, facilitating genetic studies.
Recessive lethal "balancer chromosomes" carrying visible genetic markers can be used to keep stocks of lethal alleles in a heterozygous state without recombination due to multiple inversions in the balancer.
Genetic transformation techniques have been available since 1987.
Its complete genome was sequenced and first published in 2000.
Genetic markers are commonly used in Drosophila
research, for example within balancer chromosomes or P-element inserts, and most
phenotypes are easily identifiable either with the naked eye or under a
microscope. In the list of example common markers below, the allele symbol is
followed by the name of the gene affected and a description of its phenotype.
(Note: Recessive alleles are in lower case, while dominant alleles are
Cy1: Curly; the wings curve away from the body, flight may be somewhat impaired.
e1: ebony; black body and wings (heterozygotes are also visibly darker than wild type).
Sb1: stubble; bristles are shorter and thicker than wild type.
w1: white; eyes lack pigmentation and appear white.
y1: yellow; body pigmentation and wings appear yellow. This is the fly analog of albinism.
Drosophila genes are traditionally named after the phenotype they cause when mutated. For example, the absence of a particular gene in Drosophila will result in a mutant embryo that does not develop a heart. Scientists have thus called this gene tinman, named after the Oz character of the same name. This system of nomenclature results in a wider range of gene names than in other organisms.
The genome of D. melanogaster (sequenced in 2000, and curated at the FlyBase database) contains four pairs of chromosomes: an X/Y pair, and three autosomes labeled 2, 3, and 4. The fourth chromosome is so tiny, it is often ignored, aside from its important eyeless gene. The D. melanogaster sequenced genome of 139.5 million base pairs has been annotated and contains around 15,682 genes according to Ensemble release 73. More than 60% of the genome appears to be functional non-protein-coding DNA involved in gene expression control. Determination of sex in Drosophila occurs by the X:A ratio of X chromosomes to autosomes, not because of the presence of a Y chromosome as in human sex determination. Although the Y chromosome is entirely heterochromatic, it contains at least 16 genes, many of which are thought to have male-related functions.
About 75% of known human disease genes have a recognizable match in the genome of fruit flies, and 50% of fly protein sequences have mammalian homologs. An online database called Homophila is available to search for human disease gene homologues in flies and vice versa. Drosophila is being used as a genetic model for several human diseases including the neurodegenerative disorders Parkinson's, Huntington's, spinocerebellar ataxia and Alzheimer's disease. The fly is also being used to study mechanisms underlying aging and oxidative stress, immunity, diabetes, and cancer, as well as drug abuse.
Embryogenesis in Drosophila has been extensively
studied, as its small size, short generation time, and large brood size makes it
ideal for genetic studies. It is also unique among model organisms in that
cleavage occurs in a syncytium.
D. melanogaster oogenesis
During oogenesis, cytoplasmic bridges called "ring canals" connect the forming oocyte to nurse cells. Nutrients and developmental control molecules move from the nurse cells into the oocyte. In the figure to the left, the forming oocyte can be seen to be covered by follicular support cells.
After fertilization of the oocyte, the early embryo (or syncytial embryo) undergoes rapid DNA replication and 13 nuclear divisions until about 5000 to 6000 nuclei accumulate in the unseparated cytoplasm of the embryo. By the end of the eighth division, most nuclei have migrated to the surface, surrounding the yolk sac (leaving behind only a few nuclei, which will become the yolk nuclei). After the 10th division, the pole cells form at the posterior end of the embryo, segregating the germ line from the syncytium. Finally, after the 13th division, cell membranes slowly invaginate, dividing the syncytium into individual somatic cells. Once this process is completed, gastrulation starts.
Nuclear division in the early Drosophila embryo happens so quickly, no proper checkpoints exist, so mistakes may be made in division of the DNA. To get around this problem, the nuclei that have made a mistake detach from their centrosomes and fall into the centre of the embryo (yolk sac), which will not form part of the fly.
The gene network (transcriptional and protein interactions) governing the early development of the fruit fly embryo is one of the best understood gene networks to date, especially the patterning along the anteroposterior (AP) and dorsoventral (DV) axes (See under morphogenesis).
The embryo undergoes well-characterized morphogenetic movements during gastrulation and early development, including germ-band extension, formation of several furrows, ventral invagination of the mesoderm, and posterior and anterior invagination of endoderm (gut), as well as extensive body segmentation until finally hatching from the surrounding cuticle into a first-instar larva.
During larval development, tissues known as imaginal discs grow inside the larva. Imaginal discs develop to form most structures of the adult body, such as the head, legs, wings, thorax, and genitalia. Cells of the imaginal disks are set aside during embryogenesis and continue to grow and divide during the larval stages unlike most other cells of the larva, which have differentiated to perform specialized functions and grow without further cell division. At metamorphosis, the larva forms a pupa, inside which the larval tissues are reabsorbed and the imaginal tissues undergo extensive morphogenetic movements to form adult structures.
Drosophila flies have both X and Y chromosomes, as well as autosomes. Unlike humans, the Y chromosome does not confer maleness; rather, it encodes genes necessary for making sperm. Sex is instead determined by the ratio of X chromosomes to autosomes. Furthermore, each cell "decides" whether to be male or female independently of the rest of the organism, resulting in the occasional occurrence of gynandromorphs.
Three major genes are involved in determination of
Drosophila sex. These are sex-lethal, sisterless, and deadpan. Deadpan is an
autosomal gene which inhibits sex-lethal, while sisterless is carried on the X
chromosome and inhibits the action of deadpan. An AAX cell has twice as much
deadpan as sisterless, so sex-lethal will be inhibited, creating a male.
However, an AAXX cell will produce enough sisterless to inhibit the action of
deadpan, allowing the sex-lethal gene to be transcribed to create a female.
Later, control by deadpan and sisterless disappears and what becomes important is the form of the sex-lethal gene. A secondary promoter causes transcription in both males and females. Analysis of the cDNA has shown that different forms are expressed in males and females. Sex-lethal has been shown to affect the splicing of its own mRNA. In males, the third exon is included which encodes a stop codon, causing a truncated form to be produced. In the female version, the presence of sex-lethal causes this exon to be missed out; the other seven amino acids are produced as a full peptide chain, again giving a difference between males and females.
Presence or absence of functional sex-lethal proteins now go on to affect the transcription of another protein known as doublesex. In the absence of sex-lethal, doublesex will have the fourth exon removed and be translated up to and including exon 6 (DSX-M[ale]), while in its presence the fourth exon which encodes a stop codon will produce a truncated version of the protein (DSX-F[emale]). DSX-F causes transcription of Yolk proteins 1 and 2 in somatic cells, which will be pumped into the oocyte on its production.
Unlike mammals, Drosophila flies only have innate immunity and lack an adaptive immune response. The D. melanogaster immune system can be divided into two responses: humoral and cell-mediated. The former is a systemic response mediated through the Toll and imd pathways, which are parallel systems for detecting microbes. The Toll pathway in Drosophila is known as the homologue of Toll-like pathways in mammals. Spatzle, a known ligand for the Toll pathway in flies, is produced in response to Gram-positive bacteria, parasites, and fungal infection. Upon infection, pro-Spatzle will be cleaved by protease SPE (Spatzle processing enzyme) to become active Spatzle, which then binds to the Toll receptor located on the cell surface (Fat body, hemocytes) and dimerise for activation of downstream NF-κB signaling pathways. The imd pathway, though, is triggered by Gram-negative bacteria through soluble and surface receptors (PGRP-LE and LC, respectively). D. melanogaster has a "fat body", which is thought to be homologous to the human liver. It is the primary secretory organ and produces antimicrobial peptides. These peptides are secreted into the hemolymph and bind infectious bacteria, killing them by forming pores in their cell walls. Years ago[when?] many drug companies wanted to purify these peptides and use them as antibiotics. Other than the fat body, hemocytes, the blood cells in Drosophila, are known as the homologue of mammalian monocyte/macrophages, possessing a significant role in immune responses. It is known from the literature that in response to immune challenge, hemocytes are able to secrete cytokines, for example Spatzle, to activate downstream signaling pathways in the fat body. However, the mechanism still remains unclear.
In 1971, Ron Konopka and Seymour Benzer published
"Clock mutants of Drosophila melanogaster", a paper describing the first
mutations that affected an animal's behavior. Wild-type flies show an activity
rhythm with a frequency of about a day (24 hours). They found mutants with
faster and slower rhythms, as well as broken rhythms flies that move and rest in
random spurts. Work over the following 30 years has shown that these mutations
(and others like them) affect a group of genes and their products that comprise
a biochemical or biological clock. This clock is found in a wide range of fly
cells, but the clock-bearing cells that control activity are several dozen
neurons in the fly's central brain.
Since then, Benzer and others have used behavioral screens to isolate genes involved in vision, olfaction, audition, learning/memory, courtship, pain, and other processes, such as longevity.
The first learning and memory mutants (dunce, rutabaga, etc.) were isolated by William "Chip" Quinn while in Benzer's lab, and were eventually shown to encode components of an intracellular signaling pathway involving cyclic AMP, protein kinase A, and a transcription factor known as CREB. These molecules were shown to be also involved in synaptic plasticity in Aplysia and mammals.
Male flies sing to the females during courtship using their wings to generate sound, and some of the genetics of sexual behavior have been characterized. In particular, the fruitless gene has several different splice forms, and male flies expressing female splice forms have female-like behavior and vice versa. The TRP channels nompC, nanchung, and inactive are expressed in sound-sensitive Johnston's organ neurons and participate in the transduction of sound.
Furthermore, Drosophila has been used in neuropharmacological research, including studies of cocaine and alcohol consumption. Models for Parkinson's disease also exist for flies.
The compound eye of the fruit fly contains 760 unit
eyes or ommatidia, and are one of the most advanced among insects. Each
ommatidium contains eight photoreceptor cells, support cells, pigment cells, and
a cornea. Wild-type flies have reddish pigment cells, which serve to absorb
excess blue light so the fly is not blinded by ambient light.
Each photoreceptor cell consists of two main sections, the cell body and the rhabdomere. The cell body contains the nucleus, while the 100-μm-long rhabdomere is made up of toothbrush-like stacks of membrane called microvilli. Each microvillus is 1 2 μm in length and about 60 nm in diameter. The membrane of the rhabdomere is packed with about 100 million rhodopsin molecules, the visual protein that absorbs light. The rest of the visual proteins are also tightly packed into the microvillar space, leaving little room for cytoplasm.
The photoreceptors in Drosophila express a variety of rhodopsin isoforms. The R1-R6 photoreceptor cells express rhodopsin1 (Rh1), which absorbs blue light (480 nm). The R7 and R8 cells express a combination of either Rh3 or Rh4, which absorb UV light (345 nm and 375 nm), and Rh5 or Rh6, which absorb blue (437 nm) and green (508 nm) light, respectively. Each rhodopsin molecule consists of an opsin protein covalently linked to a carotenoid chromophore, 11-cis-3-hydroxyretinal.
Expression of rhodopsin1 (Rh1) in photoreceptors R1-R6
As in vertebrate vision, visual transduction in invertebrates occurs via a G protein-coupled pathway. However, in vertebrates, the G protein is transducin, while the G protein in invertebrates is Gq (dgq in Drosophila). When rhodopsin (Rh) absorbs a photon of light its chromophore, 11-cis-3-hydroxyretinal, is isomerized to all-trans-3-hydroxyretinal. Rh undergoes a conformational change into its active form, metarhodopsin. Metarhodopsin activates Gq, which in turn activates a phospholipase Cβ (PLCβ) known as NorpA.
PLCβ hydrolyzes phosphatidylinositol (4,5)-bisphosphate (PIP2), a phospholipid found in the cell membrane, into soluble inositol triphosphate (IP3) and diacylglycerol (DAG), which stays in the cell membrane. DAG or a derivative of DAG causes a calcium-selective ion channel known as transient receptor potential (TRP) to open and calcium and sodium flows into the cell. IP3 is thought to bind to IP3 receptors in the subrhabdomeric cisternae, an extension of the endoplasmic reticulum, and cause release of calcium, but this process does not seem to be essential for normal vision.
Calcium binds to proteins such as calmodulin (CaM) and an eye-specific protein kinase C (PKC) known as InaC. These proteins interact with other proteins and have been shown to be necessary for shut off of the light response. In addition, proteins called arrestins bind metarhodopsin and prevent it from activating more Gq. A sodium-calcium exchanger known as CalX pumps the calcium out of the cell. It uses the inward sodium gradient to export calcium at a stoichiometry of 3 Na+/ 1 Ca++.
TRP, InaC, and PLC form a signaling complex by binding a scaffolding protein called InaD. InaD contains five binding domains called PDZ domain proteins, which specifically bind the C termini of target proteins. Disruption of the complex by mutations in either the PDZ domains or the target proteins reduces the efficiency of signaling. For example, disruption of the interaction between InaC, the protein kinase C, and InaD results in a delay in inactivation of the light response.
Unlike vertebrate metarhodopsin, invertebrate metarhodopsin can be converted back into rhodopsin by absorbing a photon of orange light (580 nm).
About two-thirds of the Drosophila brain is dedicated to visual processing. Although the spatial resolution of their vision is significantly worse than that of humans, their temporal resolution is around 10 times better.
The wings of a fly are capable of beating up to 220
times per second. Flies fly via straight sequences of movement interspersed by
rapid turns called saccades. During these turns, a fly is able to rotate 90 in
less than 50 milliseconds.
It was long thought[by whom?] that the characteristics of Drosophila flight were dominated by the viscosity of the air, rather than the inertia of the fly body. This view was challenged by research in the lab of Michael Dickinson, which indicated that flies perform banked turns, where the fly accelerates, slows down while turning, and accelerates again at the end of the turn, suggesting that inertia is the dominant force, as is the case with larger flying animals. However, subsequent work showed that while the viscous effects on the insect body during flight may be negligible, the aerodynamic forces on the wings themselves actually cause fruit flies' turns to be damped viscously.
Drosophila is commonly considered a pest due to its tendency to infest habitations and establishments where fruit is found; the flies may collect in homes, restaurants, stores, and other locations. Removal of an infestation can be difficult, as larvae may continue to hatch in nearby fruit even as the adult population is eliminated.
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